Monday, September 25, 2006
Nursing 1 Review Shima 402
Test Review will be done by Caralee and Fernisa at 2:30 on Tuesday (Tomorrow), September 26th. This is for your test on Thursday.
Sunday, September 24, 2006
Thursday, September 21, 2006
Issues with burning the disk
N6-Hey there, I'm having issues with burning the disk. I have successfully burned a couple, but I don't know what the deal is. So, If you would like to borrow my disk and burn one yourself or if there is someone else out there who would be willing to burn it, please feel free. Sorry! I tried!
Sunday, September 17, 2006
Some of the cardiac drugs
Hey I found a pretty decent site regarding some of this stuff. I really like things to be broken down easier than our books. Kinda like give me the meat without all the fluff.....
http://www.muhealth.org/~pharm204/cardiovascular_drugs.html
Hope it helps
Cher
http://www.muhealth.org/~pharm204/cardiovascular_drugs.html
Hope it helps
Cher
Friday, September 15, 2006
Correction N6 people
I dont know what I was thinking--There is only 1 of the N7 disks, so I don't need 2 just 1. (don't mind me, I just work here & I'm having a nervous breakdown!)
Thursday, September 07, 2006
N6 People
Hey there--I have the disks. If you would like a copy please email me at RobbyGirl1@mac.com and/or bring 2 disks to class on Monday (or whenever) and I'll burn them for you. :)
Monday, September 04, 2006
Critical Care Tutorial
Here are the Shock Key Points from the Critical Care Tutorials
1. Blood Pressure is Cardiac Output multiplied by Peripheral Resistance.
2. Cardiac Output is Heart Rate times Stroke Volume.
3. Hypotension is caused by either inadequate Cardiac Output or inadequate Peripheral Resistance
4. Heart Rate, Stroke Volume and Total Peripheral Resistance exist in dynamic equilibrium: these interactions maintain blood pressure. If one of the three becomes abnormal, the other two compensate. This represents the cardiovascular physiologic reserve.
5. Hypotension is an indication of 1) an abnormality of Heart Rate, Stroke Volume or Peripheral Resistance, & 2) failure of the others to compensate.
6. Shock is acute circulatory failure leading to inadequate tissue perfusion and end organ injury: it classified as being due to malfunction of 1) the Pump (cardiogenic), 2 ) the Tubing (distributive), or 3) the Fluid (hypovolemic).
7. The heart rate is a fundamental element of hypotension both in terms of cause (tachyarrhythmias / bradyarrhythmias) and compensation – hypotension should be accompanied by a tachycardia.
8. Low Stroke volume is caused by a problem with reception or a problem with ejection.
9. Problems with reception are: inadequate venous return or cardiac inflow obstruction.
10. Fluid loss is caused by either absolute hypovolemia (e.g. blood loss) or relative hypovolemia (“third spacing”).
11. Cardiac inflow obstruction is caused by a pericardial (tamponade) or intrathoracic process (PEEP), or a lesion within the heart itself (mitral stenosis).
12. Problems with ejection include pump failure (ischemia, overload, contusion, inflammation) and outflow obstruction (embolism, aortic stenosis, aortic crossclamps).
13. Shock caused by low peripheral vascular resistance is caused by loss of tonic vasoconstriction (vasoplegia), due to sympathectomy, anaphylaxis or sepsis, leading to relative hypovolemia.
14. Vasodilation associated with septic shock occurs due to increased synthesis of nitric oxide, activation of ATP-sensitive potassium channels in vascular smooth muscle, and deficiency of vasopressin.
1. Blood Pressure is Cardiac Output multiplied by Peripheral Resistance.
2. Cardiac Output is Heart Rate times Stroke Volume.
3. Hypotension is caused by either inadequate Cardiac Output or inadequate Peripheral Resistance
4. Heart Rate, Stroke Volume and Total Peripheral Resistance exist in dynamic equilibrium: these interactions maintain blood pressure. If one of the three becomes abnormal, the other two compensate. This represents the cardiovascular physiologic reserve.
5. Hypotension is an indication of 1) an abnormality of Heart Rate, Stroke Volume or Peripheral Resistance, & 2) failure of the others to compensate.
6. Shock is acute circulatory failure leading to inadequate tissue perfusion and end organ injury: it classified as being due to malfunction of 1) the Pump (cardiogenic), 2 ) the Tubing (distributive), or 3) the Fluid (hypovolemic).
7. The heart rate is a fundamental element of hypotension both in terms of cause (tachyarrhythmias / bradyarrhythmias) and compensation – hypotension should be accompanied by a tachycardia.
8. Low Stroke volume is caused by a problem with reception or a problem with ejection.
9. Problems with reception are: inadequate venous return or cardiac inflow obstruction.
10. Fluid loss is caused by either absolute hypovolemia (e.g. blood loss) or relative hypovolemia (“third spacing”).
11. Cardiac inflow obstruction is caused by a pericardial (tamponade) or intrathoracic process (PEEP), or a lesion within the heart itself (mitral stenosis).
12. Problems with ejection include pump failure (ischemia, overload, contusion, inflammation) and outflow obstruction (embolism, aortic stenosis, aortic crossclamps).
13. Shock caused by low peripheral vascular resistance is caused by loss of tonic vasoconstriction (vasoplegia), due to sympathectomy, anaphylaxis or sepsis, leading to relative hypovolemia.
14. Vasodilation associated with septic shock occurs due to increased synthesis of nitric oxide, activation of ATP-sensitive potassium channels in vascular smooth muscle, and deficiency of vasopressin.
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